Yes, virtually all cervical cancers are due to a high risk HPV type….many people carry more than one HPV type. There is no guarantee that you will acquire genital warts. Most people who acquire an HPV type do not show signs of abnormal cell changes or of external genital warts.
Most genital warts are due to low grade HPV types 6 or 11…however some genital warts are of a high risk HPV types. http://www.cdc.gov
Your denitist is your first resource for screening the oral area for pre-cancer lesions of the oral area.
Specific HPV type screening is rare outside of research. The most common HPV test is a screening of the cervix only…and for 13 high risk HPV types. They are about 15 high risk HPV types and 40 genital HPV types. Most often we never know what specific HPV type or types we carry. We can acquire new HPV types with a new sex partner.
Genital warts
Condylomata bearing HPV-6 or -11 have identical clinical
manifestations and histology [2]. Recent studies have shown that about
100% of GWs are caused by either HPV-6 or -11 but that 20–50% of
lesions also contain co-infections with HR HPV types [3] and [4]. GWs
do not usually result in major morbidity or mortality, but cause
significant psychological morbidity and very substantial healthcare
costs. Occasionally GWs persist for long periods of time and, rarely,
such long-standing lesions may progress to malignancy. GWs are highly
infectious, with a transmission rate of about 65% within sexual
partnerships from an infected to a susceptible sexual partner, and an
incubation period of between 3 weeks and 8 months, with the majority
developing warts at around 2–3 months [3]. Once GWs have developed,
they may show minimal change over time, become more numerous or
larger, or regress spontaneously. The majority of placebo-controlled
GW therapy trials show low rates of regression (around 5% complete
clearance) in the short term, although in one study over 16 weeks 20%
of women and 5% of men using placebo completely cleared their warts,
and 38% of women and 22% of men using placebo cleared over 50% of
their baseline warts [3]. Regressing warts contain significantly more
CD4 positive T cells, both within the stroma underlying the lesions
and the condylomata themselves, and greater expression of activation
markers [3]. There is no report of the rate of spontaneous regression
that may occur in the longer term. Following GW clearance with
therapy, recurrence is common and is often seen within 3 months in 25%
of cases, although rates of up to 67% have been observed [3]. In
clinical practice recurrences are often seen at sites of previous
lesions, and in these cases HPV infection in stem cells or
slow-turnover cells at the site of previous clearance has persisted
and then reactivated. The proportion of HPV-6/11 infections that are
either completely cleared or persist in a latent form after clinical
resolution is unknown, and, indeed, animal models suggest that both
outcomes can occur [3].
Science direct
NEW YORK (Reuters Health) Jan 10 – Human papilloma virus (HPV) is
associated
with flat penile lesions that are more common and larger in size in
partners
of women with HPV-related cervical disease, according to a report in
the
January 1st issue of the International Journal of Cancer.
Earlier studies have reported a wide spectrum of penile lesions
associated
with HPV, the authors explain, but these studies have mainly been
restricted
to sexually transmitted disease clinic patients and partners of women
with
HPV-associated disease.
HPV types 16, 18, 31, 33, and 35 are found occasionally in visible genital warts and have been associated with external genital (i.e., vulvar, penile, and anal) squamous intraepithelial neoplasia (i.e., squamous cell carcinoma in situ, bowenoid papulosis, Erythroplasia of Queyrat, or Bowen’s disease of the genitalia). These HPV types also have been associated with vaginal, anal, and CIN and anogenital and some head and neck squamous cell carcinomas. Patients who have visible genital warts are frequently infected simultaneously with multiple HPV types.
In addition to the external genitalia (i.e., penis, vulva, scrotum, perineum, and perianal skin), genital warts can occur on the uterine cervix and in the vagina, urethra, anus, and mouth. Intra-anal warts are observed predominantly in patients who have had receptive anal intercourse; these warts are distinct from perianal warts, which can occur in men and women who do not have a history of anal sex. In addition to the genital area, HPV types 6 and 11 have been associated with conjunctival, nasal, oral, and laryngeal warts. Genital warts are usually asymptomatic, but depending on the size and anatomic location, genital warts can be painful, friable, or pruritic.http://www.cdc.gov/std/treatment/2006/ge…
Many people carry multiple HPV types.
Pathology Department, Federal University of Mato Grosso do Sul
Lily L is correct. The strains of HPV that usually cause cervical cancer do not cause warts to form. They are what’s often called “a silent strain”.
However, every strain that DOES cause a wart in the cervix, does have cancer potential too. It’s just less common.
The answer to your question is NO. You are not guaranteed to get warts, even if your girlfriend had a wart forming strain. You are likely to contract the virus but it can lay dormant in your system without producing symptoms. There are no guarantees.
{ 3 comments… read them below or add one }
Yes, virtually all cervical cancers are due to a high risk HPV type….many people carry more than one HPV type. There is no guarantee that you will acquire genital warts. Most people who acquire an HPV type do not show signs of abnormal cell changes or of external genital warts.
Most genital warts are due to low grade HPV types 6 or 11…however some genital warts are of a high risk HPV types. http://www.cdc.gov
Your denitist is your first resource for screening the oral area for pre-cancer lesions of the oral area.
Specific HPV type screening is rare outside of research. The most common HPV test is a screening of the cervix only…and for 13 high risk HPV types. They are about 15 high risk HPV types and 40 genital HPV types. Most often we never know what specific HPV type or types we carry. We can acquire new HPV types with a new sex partner.
Genital warts
Condylomata bearing HPV-6 or -11 have identical clinical
manifestations and histology [2]. Recent studies have shown that about
100% of GWs are caused by either HPV-6 or -11 but that 20–50% of
lesions also contain co-infections with HR HPV types [3] and [4]. GWs
do not usually result in major morbidity or mortality, but cause
significant psychological morbidity and very substantial healthcare
costs. Occasionally GWs persist for long periods of time and, rarely,
such long-standing lesions may progress to malignancy. GWs are highly
infectious, with a transmission rate of about 65% within sexual
partnerships from an infected to a susceptible sexual partner, and an
incubation period of between 3 weeks and 8 months, with the majority
developing warts at around 2–3 months [3]. Once GWs have developed,
they may show minimal change over time, become more numerous or
larger, or regress spontaneously. The majority of placebo-controlled
GW therapy trials show low rates of regression (around 5% complete
clearance) in the short term, although in one study over 16 weeks 20%
of women and 5% of men using placebo completely cleared their warts,
and 38% of women and 22% of men using placebo cleared over 50% of
their baseline warts [3]. Regressing warts contain significantly more
CD4 positive T cells, both within the stroma underlying the lesions
and the condylomata themselves, and greater expression of activation
markers [3]. There is no report of the rate of spontaneous regression
that may occur in the longer term. Following GW clearance with
therapy, recurrence is common and is often seen within 3 months in 25%
of cases, although rates of up to 67% have been observed [3]. In
clinical practice recurrences are often seen at sites of previous
lesions, and in these cases HPV infection in stem cells or
slow-turnover cells at the site of previous clearance has persisted
and then reactivated. The proportion of HPV-6/11 infections that are
either completely cleared or persist in a latent form after clinical
resolution is unknown, and, indeed, animal models suggest that both
outcomes can occur [3].
Science direct
NEW YORK (Reuters Health) Jan 10 – Human papilloma virus (HPV) is
associated
with flat penile lesions that are more common and larger in size in
partners
of women with HPV-related cervical disease, according to a report in
the
January 1st issue of the International Journal of Cancer.
Earlier studies have reported a wide spectrum of penile lesions
associated
with HPV, the authors explain, but these studies have mainly been
restricted
to sexually transmitted disease clinic patients and partners of women
with
HPV-associated disease.
HPV types 16, 18, 31, 33, and 35 are found occasionally in visible genital warts and have been associated with external genital (i.e., vulvar, penile, and anal) squamous intraepithelial neoplasia (i.e., squamous cell carcinoma in situ, bowenoid papulosis, Erythroplasia of Queyrat, or Bowen’s disease of the genitalia). These HPV types also have been associated with vaginal, anal, and CIN and anogenital and some head and neck squamous cell carcinomas. Patients who have visible genital warts are frequently infected simultaneously with multiple HPV types.
In addition to the external genitalia (i.e., penis, vulva, scrotum, perineum, and perianal skin), genital warts can occur on the uterine cervix and in the vagina, urethra, anus, and mouth. Intra-anal warts are observed predominantly in patients who have had receptive anal intercourse; these warts are distinct from perianal warts, which can occur in men and women who do not have a history of anal sex. In addition to the genital area, HPV types 6 and 11 have been associated with conjunctival, nasal, oral, and laryngeal warts. Genital warts are usually asymptomatic, but depending on the size and anatomic location, genital warts can be painful, friable, or pruritic.http://www.cdc.gov/std/treatment/2006/ge…
Many people carry multiple HPV types.
Pathology Department, Federal University of Mato Grosso do Sul
Lily L is correct. The strains of HPV that usually cause cervical cancer do not cause warts to form. They are what’s often called “a silent strain”.
However, every strain that DOES cause a wart in the cervix, does have cancer potential too. It’s just less common.
The answer to your question is NO. You are not guaranteed to get warts, even if your girlfriend had a wart forming strain. You are likely to contract the virus but it can lay dormant in your system without producing symptoms. There are no guarantees.
The types of HPV that cause cervical cancer do not cause warts.